8 Einträge gefunden

1. PARP1 inhibition radiosensitizes HNSCC cells deficient in homologous recombination by disabling the DNA replication fork elongation response

   1. Wurster, Stephanie
   2. Hennes, Fabian
   3. Parplys, Ann C
   4. Seelbach, Jasna I
   5. Mansour Khalfallah, Wael Yassin
   6. Zielinski, Alexandra
   7. Petersen, Cordula

   2016 - Forschungsinformationssystem des UKE
2. Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1

   1. Bakr, Ali
   2. Köcher, Sabrina
   3. Volquardsen, Jennifer
   4. Petersen, Cordula
   5. Borgmann, Kerstin
   6. Dikomey, Ekkehard
   7. Rothkamm, Kai

   2016 - Forschungsinformationssystem des UKE
3. DNA repair after oncological therapy (radiotherapy and chemotherapy)

   1. Dikomey, Ekkehard
   2. Borgmann, Kerstin
   3. Kriegs, Malte
   4. Mansour Khalfallah, Wael Yassin
   5. Petersen, Cordula
   6. Rieckmann, Thorsten

   2016 - Forschungsinformationssystem des UKE
4. Involvement of ATM in homologous recombination after end resection and RAD51 nucleofilament formation

   1. Bakr, A
   2. Oing, C
   3. Köcher, S
   4. Borgmann, K
   5. Dornreiter, I
   6. Petersen, Cordula
   7. Dikomey, E

   2015 - Forschungsinformationssystem des UKE - frei zugänglich
5. High levels of RAD51 perturb DNA replication elongation and cause unscheduled origin firing due to impaired CHK1 activation

   1. Parplys, Ann Christin
   2. Seelbach, Jasna Irena
   3. Becker, Saskia
   4. Behr, Matthias
   5. Wrona, Agnieszka
   6. Jend, Camilla
   7. Mansour Khalfallah, Wael Yassin

   2015 - Forschungsinformationssystem des UKE
6. Inhibition of PARP1-dependent end-joining contributes to Olaparib-mediated radiosensitization in tumor cells

   1. Kötter, Annika
   2. Cornils, Kerstin
   3. Borgmann, Kerstin
   4. Dahm-Daphi, Jochen
   5. Petersen, Cordula
   6. Dikomey, Ekkehard
   7. Mansour Khalfallah, Wael Yassin

   2014 - Forschungsinformationssystem des UKE - frei zugänglich
7. The absence of Ku but not defects in classical non-homologous end-joining is required to trigger PARP1-dependent end-joining

   1. Mansour Khalfallah, Wael Yassin
   2. Borgmann, K
   3. Petersen, Cordula
   4. Dikomey, Ekkehard
   5. Dahm-Daphi, Jochen

   2013 - Forschungsinformationssystem des UKE
8. Aberrant overexpression of miR-421 downregulates ATM and leads to a pronounced DSB repair defect and clinical hypersensitivity in SKX squamous cell carcinoma.

   1. Mansour Khalfallah, Wael Yassin
   2. Bogdanova, Natalia V
   3. Kasten-Pisula, Ulla
   4. Rieckmann, Thorsten
   5. Köcher, Sabrina
   6. Borgmann, Kerstin
   7. Baumann, Michael

   2013 - Forschungsinformationssystem des UKE