6 Einträge gefunden

1. Activation of autophagy ameliorates cardiomyopathy in Mybpc3-targeted knock-in mice

   1. Singh, Sonia
   2. Zech, Antonia Theresa Luisa
   3. Geertz, Birgit
   4. Reischmann, Silke
   5. Osinska, Hanna
   6. Prondzynski, Maksymilian
   7. Krämer, Elisabeth

   2017 - Forschungsinformationssystem des UKE
2. Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice

   1. Flenner, Frederik
   2. Geertz, Birgit
   3. Reischmann, Silke
   4. Weinberger, Florian
   5. Eschenhagen, Thomas
   6. Carrier, Lucie
   7. Friedrich, Felix

   2017 - Forschungsinformationssystem des UKE - frei zugänglich
3. Ranolazine antagonizes catecholamine-induced dysfunction in isolated cardiomyocytes, but lacks long-term therapeutic effects in vivo in a mouse model of hypertrophic cardiomyopathy

   1. Flenner, Frederik
   2. Friedrich, Felix W
   3. Ungeheuer, Nele
   4. Christ, Torsten
   5. Geertz, Birgit
   6. Reischmann, Silke
   7. Wagner, Stefan

   2016 - Forschungsinformationssystem des UKE
4. I-1-deficiency negatively impacts survival in a cardiomyopathy mouse model

   1. Friedrich, Felix
   2. Sotoud, Hannieh
   3. Geertz, Birgit
   4. Weber, Silvio
   5. Flenner, Frederik
   6. Reischmann, Silke
   7. Eschenhagen, Thomas

   2015 - Forschungsinformationssystem des UKE - frei zugänglich
5. Proteasome inhibition slightly improves cardiac function in mice with hypertrophic cardiomyopathy

   1. Schlossarek, Saskia
   2. Singh, Sonia R
   3. Geertz, Birgit
   4. Schulz, Herbert
   5. Reischmann, Silke
   6. Hübner, Norbert
   7. Carrier, Lucie

   2014 - Forschungsinformationssystem des UKE - frei zugänglich
6. Rescue of cardiomyopathy through U7snRNA-mediated exon skipping in Mybpc3-targeted knock-in mice

   1. Hornung, Christina
   2. Behrens, Verena
   3. Reischmann, Silke
   4. Geertz, Birgit
   5. Stimpel, Doreen
   6. Weinberger, Florian
   7. Schlossarek, Saskia

   2013 - Forschungsinformationssystem des UKE - frei zugänglich