12 Einträge gefunden

1. Establishment of a Transformation Coupled in vitro End Joining Assay to Estimate Radiosensitivity in Tumor Cells

   1. Degenhardt, Sarah
   2. Dreffke, Kristin
   3. Schötz, Urlike
   4. Petersen, Cordula
   5. Engenhart-Cabillic, Rita
   6. Rothkamm, Kai
   7. Dahm-Daphi, Jochen

   2020 - Forschungsinformationssystem des UKE
2. Inhibition of PARP1-dependent end-joining contributes to Olaparib-mediated radiosensitization in tumor cells

   1. Kötter, Annika
   2. Cornils, Kerstin
   3. Borgmann, Kerstin
   4. Dahm-Daphi, Jochen
   5. Petersen, Cordula
   6. Dikomey, Ekkehard
   7. Mansour Khalfallah, Wael Yassin

   2014 - Forschungsinformationssystem des UKE - frei zugänglich
3. ATM is required for the repair of Topotecan-induced replication-associated double-strand breaks

   1. Köcher, Sabrina
   2. Spies-Naumann, Anja
   3. Kriegs, Malte
   4. Dahm-Daphi, Jochen
   5. Dornreiter, Irena

   2013 - Forschungsinformationssystem des UKE
4. p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation.

   1. Rieckmann, Thorsten
   2. Kriegs, Malte
   3. Nitsch, Lena
   4. Hoffer, Konstantin
   5. Rohaly, G
   6. Köcher, Sabrina
   7. Petersen, Cordula

   2013 - Forschungsinformationssystem des UKE
5. The absence of Ku but not defects in classical non-homologous end-joining is required to trigger PARP1-dependent end-joining

   1. Mansour Khalfallah, Wael Yassin
   2. Borgmann, K
   3. Petersen, Cordula
   4. Dikomey, Ekkehard
   5. Dahm-Daphi, Jochen

   2013 - Forschungsinformationssystem des UKE
6. Radiation-induced double-strand breaks require ATM but not Artemis for homologous recombination during S-phase.

   1. Köcher, Sabrina
   2. Rieckmann, Thorsten
   3. Rohaly, Gabor
   4. Mansour, Wael
   5. Dikomey, Ekkehard
   6. Dornreiter, Irena
   7. Dahm-Daphi, Jochen

   2012 - Forschungsinformationssystem des UKE - frei zugänglich
7. Cisplatin resistance induced in germ cell tumour cells is due to reduced susceptibility towards cell death but not to altered DNA damage induction or repair.

   1. Fenske, Annabelle E
   2. Glaesener, Stephanie
   3. Bokemeyer, Carsten
   4. Thomale, Juergen
   5. Dahm-Daphi, Jochen
   6. Honecker, Friedemann
   7. Dartsch, Dorothee C

   2012 - Forschungsinformationssystem des UKE
8. Cisplatin resistance induced in germ cell tumour cells is due to reduced susceptibility towards cell death but not to altered DNA damage induction or repair

   1. Fenske, Annabelle E.
   2. Glaesener, Stephanie
   3. Bokemeyer, Carsten
   4. Thomale, Jürgen
   5. Dahm-Daphi, Jochen
   6. Honecker, Friedemann
   7. Dartsch, Dorothee

   2012 - Forschungsinformationssystem der UHH
9. EGF receptor inhibition radiosensitizes NSCLC cells by inducing senescence in cells sustaining DNA double-strand breaks.

   1. Wang, Meng
   2. Morsbach, Fabian
   3. Sander, David
   4. Gheorghiu, Liliana
   5. Nanda, Akash
   6. Benes, Cyril
   7. Kriegs, Malte

   2011 - Forschungsinformationssystem des UKE
10. FANCD2 but not FANCA promotes cellular resistance to type II topoisomerase poisons.

    1. Kachnic, Lisa A
    2. Li, Li
    3. Fournier, Loreen
    4. Ferraiolo, Natalie
    5. Dahm-Daphi, Jochen
    6. Borgmann, Kerstin
    7. Willers, Henning

    2011 - Forschungsinformationssystem des UKE
11. In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.

    1. Myllynen, Laura
    2. Rieckmann, Thorsten
    3. Dahm-Daphi, Jochen
    4. Kasten-Pisula, Ulla
    5. Petersen, Cordula
    6. Dikomey, Ekkehard
    7. Kriegs, Malte

    2011 - Forschungsinformationssystem des UKE
12. ATR-p53 restricts homologous recombination in response to replicative stress but does not limit DNA interstrand crosslink repair in lung cancer cells.

    1. Sirbu, Bianca M
    2. Lachmayer, Sarah J.
    3. Wülfing, Verena
    4. Marten, Lara M.
    5. Clarkson, Katie E
    6. Lee, Linda W
    7. Gheorghiu, Liliana

    2011 - Forschungsinformationssystem des UKE - frei zugänglich