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The sheddase ADAM10 is a potent modulator of prion disease
2015 - Forschungsinformationssystem des UKE - frei zugänglich
Postnatal disruption of the disintegrin/metalloproteinase ADAM10 in brain causes epileptic seizures, learning deficits, altered spine morphology, and defective synaptic functions
2013 - Forschungsinformationssystem des UKE
Lack of a-disintegrin-and-metalloproteinase ADAM10 leads to intracellular accumulation and loss of shedding of the cellular prion protein in vivo.
2011 - Forschungsinformationssystem des UKE - frei zugänglich
The disintegrin/metalloproteinase ADAM10 is essential for the establishment of the brain cortex.
2010 - Forschungsinformationssystem des UKE