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19 Einträge gefunden

Impaired Fear Extinction Due to a Deficit in Ca(2+) Influx Through L-Type Voltage-Gated Ca(2+) Channels in Mice Deficient for Tenascin-C
2017 - Forschungsinformationssystem des UKE - frei zugänglich
Age-dependent loss of parvalbumin-expressing hippocampal interneurons in mice deficient in CHL1, a mental retardation and schizophrenia susceptibility gene.
2015 - Forschungsinformationssystem des UKE
Tenascin-R promotes assembly of the extracellular matrix of perineuronal nets via clustering of aggrecan.
2014 - Forschungsinformationssystem des UKE
Restoration of synaptic plasticity and learning in young and aged NCAM-deficient mice by enhancing neurotransmission mediated by GluN2A-containing NMDA receptors.
2012 - Forschungsinformationssystem des UKE
Neural cell adhesion molecule ablation in mice causes hippocampal dysplasia and loss of septal cholinergic neurons.
2011 - Forschungsinformationssystem des UKE
Transgenic mice overexpressing the extracellular domain of NCAM are impaired in working memory and cortical plasticity.
2011 - Forschungsinformationssystem des UKE
Improved reversal learning and working memory and enhanced reactivity to novelty in mice with enhanced GABAergic innervation in the dentate gyrus.
2010 - Forschungsinformationssystem des UKE
Functional consequences of the interactions among the neural cell adhesion molecule NCAM, the receptor tyrosine kinase TrkB, and the inwardly rectifying K+ channel KIR3.3.
2010 - Forschungsinformationssystem des UKE
The 14-3-3 protein binds to the cell adhesion molecule L1, promotes L1 phosphorylation by CKII and influences L1-dependent neurite outgrowth.
2010 - Forschungsinformationssystem des UKE - frei zugänglich
NCAM-induced neurite outgrowth depends on binding of calmodulin to NCAM and on nuclear import of NCAM and fak fragments.
2010 - Forschungsinformationssystem des UKE
Retarded kindling progression in mice deficient in the extracellular matrix glycoprotein tenascin-R.
2009 - Forschungsinformationssystem des UKE
Genetic ablation of tenascin-C expression leads to abnormal hippocampal CA1 structure and electrical activity in vivo.
2009 - Forschungsinformationssystem des UKE
Modulation of synaptic transmission and plasticity by cell adhesion and repulsion molecules.
2008 - Forschungsinformationssystem des UKE
Fibroblast growth factor-regulated palmitoylation of the neural cell adhesion molecule determines neuronal morphogenesis.
2008 - Forschungsinformationssystem des UKE
Hippocampal metaplasticity induced by deficiency in the extracellular matrix glycoprotein tenascin-R.
2007 - Forschungsinformationssystem des UKE
Activity-dependent formation and functions of chondroitin sulfate-rich extracellular matrix of perineuronal nets.
2007 - Forschungsinformationssystem des UKE
Reduced reactivity to novelty, impaired social behavior, and enhanced basal synaptic excitatory activity in perforant path projections to the dentate gyrus in young adult mice deficient in the neural cell adhesion molecule CHL1.
2007 - Forschungsinformationssystem des UKE
Morphogenic signaling in neurons via neurotransmitter receptors and small GTPases.
2007 - Forschungsinformationssystem des UKE
Enhanced perisomatic inhibition and impaired long-term potentiation in the CA1 region of juvenile CHL1-deficient mice.
2006 - Forschungsinformationssystem des UKE

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Impaired Fear Extinction Due to a Deficit in Ca(2+) Influx Through L-Type Voltage-Gated Ca(2+) Channels in Mice Deficient for Tenascin-C

Age-dependent loss of parvalbumin-expressing hippocampal interneurons in mice deficient in CHL1, a mental retardation and schizophrenia susceptibility gene.

Tenascin-R promotes assembly of the extracellular matrix of perineuronal nets via clustering of aggrecan.

Restoration of synaptic plasticity and learning in young and aged NCAM-deficient mice by enhancing neurotransmission mediated by GluN2A-containing NMDA receptors.

Neural cell adhesion molecule ablation in mice causes hippocampal dysplasia and loss of septal cholinergic neurons.

Transgenic mice overexpressing the extracellular domain of NCAM are impaired in working memory and cortical plasticity.

Improved reversal learning and working memory and enhanced reactivity to novelty in mice with enhanced GABAergic innervation in the dentate gyrus.

Functional consequences of the interactions among the neural cell adhesion molecule NCAM, the receptor tyrosine kinase TrkB, and the inwardly rectifying K+ channel KIR3.3.

The 14-3-3 protein binds to the cell adhesion molecule L1, promotes L1 phosphorylation by CKII and influences L1-dependent neurite outgrowth.

NCAM-induced neurite outgrowth depends on binding of calmodulin to NCAM and on nuclear import of NCAM and fak fragments.

Retarded kindling progression in mice deficient in the extracellular matrix glycoprotein tenascin-R.

Genetic ablation of tenascin-C expression leads to abnormal hippocampal CA1 structure and electrical activity in vivo.

Modulation of synaptic transmission and plasticity by cell adhesion and repulsion molecules.

Fibroblast growth factor-regulated palmitoylation of the neural cell adhesion molecule determines neuronal morphogenesis.

Hippocampal metaplasticity induced by deficiency in the extracellular matrix glycoprotein tenascin-R.

Activity-dependent formation and functions of chondroitin sulfate-rich extracellular matrix of perineuronal nets.

Reduced reactivity to novelty, impaired social behavior, and enhanced basal synaptic excitatory activity in perforant path projections to the dentate gyrus in young adult mice deficient in the neural cell adhesion molecule CHL1.

Morphogenic signaling in neurons via neurotransmitter receptors and small GTPases.

Enhanced perisomatic inhibition and impaired long-term potentiation in the CA1 region of juvenile CHL1-deficient mice.