Suchergebnis | Hamburg Open Science

Facetten

16 Einträge gefunden

Src family kinase targeting in head and neck tumor cells using SU6656, PP2 and dasatinib
2023 - Forschungsinformationssystem des UKE - frei zugänglich
Effiziente DNA-Reparatur vermindert Replikationsstress und führt zu weniger immunogener zytosolischer DNA in radioresistenten Brustkrebsstammzellen, Efficient DNA repair mitigates replication stress resulting in less immunogenic cytosolic DNA in radioresistant breast cancer stem cells
2022 - Forschungsinformationssystem des UKE - frei zugänglich
Dual Inhibition of PARP and the Intra-S/G2 Cell Cycle Checkpoints Results in Highly Effective Radiosensitization of HPV-Positive HNSCC Cells
2021 - Forschungsinformationssystem des UKE - frei zugänglich
DNA Damage Response during Replication Correlates with CIN70 Score and Determines Survival in HNSCC Patients
2021 - Forschungsinformationssystem des UKE - frei zugänglich
Establishment of a Transformation Coupled in vitro End Joining Assay to Estimate Radiosensitivity in Tumor Cells
2020 - Forschungsinformationssystem des UKE
Exploiting Chromosomal Instability of PTEN-Deficient Triple-Negative Breast Cancer Cell Lines for the Sensitization against PARP1 Inhibition in a Replication-Dependent Manner
2020 - Forschungsinformationssystem des UKE
Radiosensitisation and enhanced tumour growth delay of colorectal cancer cells by sustained treatment with trifluridine/tipiracil and X-rays
2020 - Forschungsinformationssystem des UKE
Receptor tyrosine kinase MET as potential target of multi-kinase inhibitor and radiosensitizer sorafenib in HNSCC
2019 - Forschungsinformationssystem des UKE
BCL2-overexpressing prostate cancer cells rely on PARP1-dependent end-joining and are sensitive to combined PARP inhibitor and radiation therapy
2018 - Forschungsinformationssystem des UKE
G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1
2017 - Forschungsinformationssystem des UKE
Analyzing the influence of kinase inhibitors on DNA repair by differential proteomics of chromatin-interacting proteins and nuclear phospho-proteins
2017 - Forschungsinformationssystem des UKE
Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1
2016 - Forschungsinformationssystem des UKE
Similar cisplatin sensitivity of HPV-positive and -negative HNSCC cell lines
2016 - Forschungsinformationssystem des UKE
Sorafenib inhibits cell growth but fails to enhance radio- and chemosensitivity of glioblastoma cell lines
2016 - Forschungsinformationssystem des UKE
EGFRvIII does not affect radiosensitivity with or without gefitinib treatment in glioblastoma cells
2015 - Forschungsinformationssystem des UKE
Quantitative proteomics unveiled: Regulation of DNA double strand break repair by EGFR involves PARP1
2015 - Forschungsinformationssystem des UKE

Aktive Facetten

Facetten

Zugriff

Einrichtung

Medientyp

Karte

Erscheinungsjahr

Autor/in

Sprache

Src family kinase targeting in head and neck tumor cells using SU6656, PP2 and dasatinib

Effiziente DNA-Reparatur vermindert Replikationsstress und führt zu weniger immunogener zytosolischer DNA in radioresistenten Brustkrebsstammzellen, Efficient DNA repair mitigates replication stress resulting in less immunogenic cytosolic DNA in radioresistant breast cancer stem cells

Dual Inhibition of PARP and the Intra-S/G2 Cell Cycle Checkpoints Results in Highly Effective Radiosensitization of HPV-Positive HNSCC Cells

DNA Damage Response during Replication Correlates with CIN70 Score and Determines Survival in HNSCC Patients

Establishment of a Transformation Coupled in vitro End Joining Assay to Estimate Radiosensitivity in Tumor Cells

Exploiting Chromosomal Instability of PTEN-Deficient Triple-Negative Breast Cancer Cell Lines for the Sensitization against PARP1 Inhibition in a Replication-Dependent Manner

Radiosensitisation and enhanced tumour growth delay of colorectal cancer cells by sustained treatment with trifluridine/tipiracil and X-rays

Receptor tyrosine kinase MET as potential target of multi-kinase inhibitor and radiosensitizer sorafenib in HNSCC

BCL2-overexpressing prostate cancer cells rely on PARP1-dependent end-joining and are sensitive to combined PARP inhibitor and radiation therapy

G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1

Analyzing the influence of kinase inhibitors on DNA repair by differential proteomics of chromatin-interacting proteins and nuclear phospho-proteins

Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1

Similar cisplatin sensitivity of HPV-positive and -negative HNSCC cell lines

Sorafenib inhibits cell growth but fails to enhance radio- and chemosensitivity of glioblastoma cell lines

EGFRvIII does not affect radiosensitivity with or without gefitinib treatment in glioblastoma cells

Quantitative proteomics unveiled: Regulation of DNA double strand break repair by EGFR involves PARP1