Suchergebnis | Hamburg Open Science

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18 Einträge gefunden

G2 checkpoint targeting via Wee1 inhibition radiosensitizes EGFRvIII-positive glioblastoma cells
2023 - Forschungsinformationssystem des UKE - frei zugänglich
Src family kinase targeting in head and neck tumor cells using SU6656, PP2 and dasatinib
2023 - Forschungsinformationssystem des UKE - frei zugänglich
Dual Inhibition of PARP and the Intra-S/G2 Cell Cycle Checkpoints Results in Highly Effective Radiosensitization of HPV-Positive HNSCC Cells
2021 - Forschungsinformationssystem des UKE - frei zugänglich
Radiosensitisation and enhanced tumour growth delay of colorectal cancer cells by sustained treatment with trifluridine/tipiracil and X-rays
2020 - Forschungsinformationssystem des UKE
Analyzing expression and phosphorylation of the EGF receptor in HNSCC
2019 - Forschungsinformationssystem des UKE - frei zugänglich
G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1
2017 - Forschungsinformationssystem des UKE
Similar cisplatin sensitivity of HPV-positive and -negative HNSCC cell lines
2016 - Forschungsinformationssystem des UKE
Effect of sorafenib on cisplatin-based chemoradiation in head and neck cancer cells
2016 - Forschungsinformationssystem des UKE
DNA repair after oncological therapy (radiotherapy and chemotherapy)
2016 - Forschungsinformationssystem des UKE
Radiosensitization of HNSCC cells by EGFR inhibition depends on the induction of cell cycle arrests
2016 - Forschungsinformationssystem des UKE
EGFRvIII does not affect radiosensitivity with or without gefitinib treatment in glioblastoma cells
2015 - Forschungsinformationssystem des UKE
The inhibition of PARP but not EGFR results in the radiosensitization of HPV/p16-positive HNSCC cell lines
2014 - Forschungsinformationssystem des UKE
HNSCC cell lines positive for HPV and p16 possess higher cellular radiosensitivity due to an impaired DSB repair capacity.
2013 - Forschungsinformationssystem des UKE
Aberrant overexpression of miR-421 downregulates ATM and leads to a pronounced DSB repair defect and clinical hypersensitivity in SKX squamous cell carcinoma.
2013 - Forschungsinformationssystem des UKE
HPV-positive HNSCC cell lines but not primary human fibroblasts are radiosensitized by the inhibition of Chk1
2013 - Forschungsinformationssystem des UKE
p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation.
2013 - Forschungsinformationssystem des UKE
In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.
2011 - Forschungsinformationssystem des UKE
Impact of HPV status on treatment of squamous cell cancer of the oropharynx: what we know and what we need to know.
2011 - Forschungsinformationssystem des UKE

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G2 checkpoint targeting via Wee1 inhibition radiosensitizes EGFRvIII-positive glioblastoma cells

Src family kinase targeting in head and neck tumor cells using SU6656, PP2 and dasatinib

Dual Inhibition of PARP and the Intra-S/G2 Cell Cycle Checkpoints Results in Highly Effective Radiosensitization of HPV-Positive HNSCC Cells

Radiosensitisation and enhanced tumour growth delay of colorectal cancer cells by sustained treatment with trifluridine/tipiracil and X-rays

Analyzing expression and phosphorylation of the EGF receptor in HNSCC

G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1

Similar cisplatin sensitivity of HPV-positive and -negative HNSCC cell lines

Effect of sorafenib on cisplatin-based chemoradiation in head and neck cancer cells

DNA repair after oncological therapy (radiotherapy and chemotherapy)

Radiosensitization of HNSCC cells by EGFR inhibition depends on the induction of cell cycle arrests

EGFRvIII does not affect radiosensitivity with or without gefitinib treatment in glioblastoma cells

The inhibition of PARP but not EGFR results in the radiosensitization of HPV/p16-positive HNSCC cell lines

HNSCC cell lines positive for HPV and p16 possess higher cellular radiosensitivity due to an impaired DSB repair capacity.

Aberrant overexpression of miR-421 downregulates ATM and leads to a pronounced DSB repair defect and clinical hypersensitivity in SKX squamous cell carcinoma.

HPV-positive HNSCC cell lines but not primary human fibroblasts are radiosensitized by the inhibition of Chk1

p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation.

In tumor cells regulation of DNA double strand break repair through EGF receptor involves both NHEJ and HR and is independent of p53 and K-Ras status.

Impact of HPV status on treatment of squamous cell cancer of the oropharynx: what we know and what we need to know.