14 Einträge gefunden

1. Sacubitrilat reduces pro-arrhythmogenic sarcoplasmic reticulum Ca2+ leak in human ventricular cardiomyocytes of patients with end-stage heart failure

   1. Eiringhaus, Jörg
   2. Wünsche, Christoph M
   3. Tirilomis, Petros
   4. Herting, Jonas
   5. Bork, Nadja
   6. Nikolaev, Viacheslav O
   7. Hasenfuss, Gerd

   2020 - Forschungsinformationssystem des UKE
2. Mechanistic role of the CREB-regulated transcription coactivator 1 in cardiac hypertrophy

   1. Morhenn, Karoline
   2. Quentin, Thomas
   3. Wichmann, Helen
   4. Steinmetz, Michael
   5. Prondzynski, Maksymilian
   6. Söhren, Klaus-Dieter
   7. Christ, Torsten

   2019 - Forschungsinformationssystem des UKE
3. Mechanistic role of the CREB-regulated transcription coactivator 1 in cardiac hypertrophy

   1. Morhenn, Karoline
   2. Quentin, Thomas
   3. Wichmann, Helen
   4. Steinmetz, Michael
   5. Prondzynski, Maksymilian
   6. Söhren, Klaus Dieter
   7. Christ, Torsten

   2019 - Forschungsinformationssystem der UHH
4. The functional consequences of sodium channel NaV1.8 in human left ventricular hypertrophy

   1. Ahmad, Shakil
   2. Tirilomis, Petros
   3. Pabel, Steffen
   4. Dybkova, Nataliya
   5. Hartmann, Nico
   6. Molina, Cristina E.
   7. Tirilomis, Theodoros

   2019 - Forschungsinformationssystem des UKE - frei zugänglich
5. In vivo model with targeted cAMP biosensor reveals changes in receptor-microdomain communication in cardiac disease

   1. Sprenger, Julia U
   2. Perera, Ruwan K
   3. Steinbrecher, Julia H
   4. Lehnart, Stephan E
   5. Maier, Lars S
   6. Hasenfuss, Gerd
   7. Nikolaev, Viacheslav O

   2015 - Forschungsinformationssystem des UKE - frei zugänglich
6. Summative assessments are more powerful drivers of student learning than resource intensive teaching formats

   1. Raupach, Tobias
   2. Brown, Jamie
   3. Anders, Sven
   4. Hasenfuss, Gerd
   5. Harendza, Sigrid

   2013 - Forschungsinformationssystem des UKE - frei zugänglich
7. Impaired physical quality of life in patients with diastolic dysfunction associates more strongly with neurohumoral activation than with echocardiographic parameters: quality of life in diastolic dysfunction.

   1. Edelmann, Frank
   2. Stahrenberg, Raoul
   3. Polzin, Friederike
   4. Kockskämper, Anke
   5. Düngen, Hans-Dirk
   6. Duvinage, André
   7. Binder, Lutz

   2011 - Forschungsinformationssystem des UKE
8. Constitutively active phosphatase inhibitor-1 improves cardiac contractility in young mice but is deleterious after catecholaminergic stress and with aging.

   1. Wittköpper, Katrin
   2. Fabritz, Larissa
   3. Neef, Stefan
   4. Ort, Katharina R
   5. Grefe, Clemens
   6. Unsöld, Bernhard
   7. Kirchhof, Paulus

   2010 - Forschungsinformationssystem des UKE
9. Effects of "minimally invasive curricular surgery" - a pilot intervention study to improve the quality of bedside teaching in medical education.

   1. Raupach, Tobias
   2. Anders, Sven
   3. Pukrop, Tobias
   4. Hasenfuss, Gerd
   5. Harendza, Sigrid

   2009 - Forschungsinformationssystem des UKE
10. Phosphatase inhibitor-1-deficient mice are protected from catecholamine-induced arrhythmias and myocardial hypertrophy.

    1. El-Armouche, Ali
    2. Wittköpper, Katrin
    3. Degenhardt, Franziska
    4. Weinberger, Florian
    5. Didié, Michael
    6. Melnychenko, Ivan
    7. Grimm, Michael

    2008 - Forschungsinformationssystem des UKE
11. Influence of pyruvate on contractile performance and Ca(2+) cycling in isolated failing human myocardium.

    1. Hasenfuss, Gerd
    2. Maier, Lars S
    3. Hermann, Hans-Peter
    4. Lüers, Claus
    5. Hünlich, Mark
    6. Zeitz, Oliver
    7. Janssen, Paul M L

    2002 - Forschungsinformationssystem des UKE
12. Potentiation of beta-adrenergic inotropic response by pyruvate in failing human myocardium.

    1. Hermann, Hans Peter
    2. Zeitz, Oliver
    3. Lehnart, Stephan E
    4. Keweloh, Boris
    5. Datz, Nicolin
    6. Hasenfuss, Gerd
    7. Janssen, Paul M L

    2002 - Forschungsinformationssystem des UKE
13. Hydroxyl radical-induced acute diastolic dysfunction is due to calcium overload via reverse-mode Na(+)-Ca(2+) exchange.

    1. Zeitz, Oliver
    2. Maass, A Eveline
    3. Phuc, Van Nguyen
    4. Hensmann, Geerd
    5. Kögler, Harald
    6. Möller, Karsten
    7. Hasenfuss, Gerd

    2002 - Forschungsinformationssystem des UKE
14. Intracellular beta-blockade: overexpression of Galpha(i2) depresses the beta-adrenergic response in intact myocardium.

    1. Janssen, Paul M L
    2. Schillinger, Wolfgang
    3. Donahue, J Kevin
    4. Zeitz, Oliver
    5. Emami, Shahriyar
    6. Lehnart, Stephan E
    7. Weil, Joachim

    2002 - Forschungsinformationssystem des UKE